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The effects of spinal administration of atropine (0.4 mg/kg, sc) on the gene expression of preproenkephalin (ppENK) were investigated by in situ hybridization in the rat. Intense immunoreactivity for ppENK was observed in the dorsal horn of the spinal cord. Atropine treatment significantly reduced the number of ppENK mRNA-positive neurons in laminae I and II of the spinal dorsal horn, but not those in laminae III-V. In the ventral horn, however, the atropine treatment did not affect ppENK expression. These results suggest that the central cholinergic system is involved in the regulation of ppENK expression in the spinal cord.
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